A controlled postoperative intravenous study did not demonstrate significance for its key meal-tolerance endpoint. Its population, route and purpose differ from community recovery use.
A negative result in that indication does not prove universal inactivity. It also does not validate subcutaneous performance stacks. One source contains an apparent dose-unit error that must not be silently corrected.
Sources and proposed-mechanism explanation
The proposed rationale is stimulation of GH release. A hormonal response and a meaningful clinical outcome are separate questions.